Glypican 3 specific T cell receptors for adoptive T cell therapy

Cáncer

Target: GPC3 (Glypican 3)

  • Introduction:
    Identification of different TCR clonotypes specific for an HLA-A2-restricted epitope of GPC3. Cloning and application of these TCRs in the treatment of hepatocarcinoma.

  • Results:
    The developed TCRs recognize and eliminate HLA-A2+GPC3+ tumor cells and have great potential for engineering the patient's autologous T cells to treat GPC3+ tumors.

  • Keywords:
  • Cáncer
  • ,
  • Terapia celular
  • ,
  • Hepatocarcinoma
  • Early Discovery
  • Discovery
  • Preclinical
  • Clinical
  • Diagnostics

Approach

  • Our laboratory has identified different TCR clonotypes specific for an HLA-A2-restricted epitope of GPC3.
  • TCRs have been cloned and primary human T cells have been genetically modified with amphotropic retrovirus coding for these TCRs.
  • We have analyzed the capacity of GPC3-TCR redirected T cells to recognize and kill HEPG2 hepatoblastoma tumor cells (naturally HLA-A2+GPC3+).
  • These data support the hypothesis that the loss of PLA2G4E might play a key role in AD onset and/or progression.

Key concepts and Target Identification

  • Primary human T cells expressing GPC3 TCRs specifically recognize HEPG2 tumor cells, exhibiting intense upregulation of the activation-dependent molecule CD137. They also proliferate and produce effector cytokines.
  • Importantly, human T cells expressing GPC3 TCRs specifically kill HEPG2 tumor cells in a major histocompatibility complex (MHC) class I-dependent manner.
  • Certain preferred TCRs, such as TCR-6, demonstrate excellent effector functions against HEPG2 tumor cells.

Target Validation

  • In vitro experiments demonstrated that human T cells genetically engineered to express GPC3-specific TCRs are able to recognize and kill HLA-A2+GPC3+ tumor cells.
  • These TCRs have a great potential to engineer patient's autologous T cells to treat GPC3+ tumors.

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